The study identified over 1,000 genes that are responsible for a severe form of coronavirus infection. It was conducted by scientists from the University of Sheffield in the UK and Stanford University in the USA.
Over the past two years, it has become absolutely clear that COVID-19 affects different people in different ways. Since the start of the pandemic in the winter of 2020, it has been clear that advanced age and the presence of some chronic diseases increase the risk of severe coronavirus and death due to it. That is why older people sought to be more securely isolated until the advent of a vaccine. But as the pandemic has evolved, other features have become visible. Not every diabetic or elderly person felt ill with the development of coronavirus, some had only mild symptoms even when they ingested the deadly version of the Delta strain.
But younger and seemingly healthy patients faced precisely the most severe form of coronavirus, which led to death within a few days. The reason for such a diverse effect of the disease on the body has always interested scientists. And now, more than 1,000 genes have been identified that are associated with the development of a severe form of COVID-19. Many of them specifically affect the functionality of the so-called natural “killer cells” – the main soldiers of our immune system, which come to the body's defense in extreme cases, attacking virus-infected cells and suppressing cancer at an early stage.
The findings of this study are very significant, as they provide strong evidence that some people have an individual or genetic predisposition to a more severe form of coronavirus. Therefore, there are also hereditary risks in addition to purely physiological ones, such as age and concomitant diseases. And this brings humanity closer to the development of more personalized medicines to fight COVID-19. (READ MORE) 
Stanford University
Medicine
refers to among the most prestigious institutions of higher education not only in the United States of America, but throughout the world