The study showed that the cause of Alzheimer's disease is not at all an increase in the amount of beta-amyloid proteins, but its reduction. This conclusion was made by researchers from the University of Cincinnati.
American scientists have questioned the prevailing theory about the origin of Alzheimer's disease. It is believed that this incurable disease occurs due to the accumulation of beta-amyloid proteins in the brain, which are woven into plaques and prevent neurons from communicating with each other. And now, researchers from Cincinnati have come to the exact opposite conclusion, which could overturn the generally accepted concept of senile dementia, considered for the past 100 years.
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Researchers have suggested that plaques in the brain called amyloid plaques are actually the result of falling levels of beta-amyloids in the brain, not growth. And this drop occurs due to the fact that a normal protein under conditions of biological, metabolic or infectious stress is transformed into those very abnormal plaques.
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The paradox is that many people accumulate plaques in the brain over the years, but not all of them give rise to senile dementia. However, these plaques remain the focus of research, as they are the focus of most experimental drugs. But what should be fought is not with a decrease in amyloid plaques in the brain, but with the fact that the amount of beta-amyloid proteins does not fall. It is the decrease in the level of the soluble form of the protein that can be toxic, as the study showed. (READ MORE) 
University of Cincinnati
Medicine
public (government) science research university located in Cincinnati, Ohio