In experiments with laboratory mice and healthy humans, intense exercise increased the level of the ATP7A protein, which can trigger the process of angiogenesis. And this is very important for repairing damage in diabetes.
Angiogenesis is the ability to form new blood vessels. In victims of diabetes, not only existing vessels are damaged, but the innate ability to grow new ones is also impaired. And all this threatens with very serious complications. Researchers in vascular biology at the Medical College of Georgia have proven that regular training is required to counter this. They provided the first evidence that even one 45-minute session of moderate-intensity exercise allowed more submicroscopic exosomes filled with biologically active cargo to deliver more ATP7A protein directly to cells.
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There are other more sophisticated and efficient delivery services for this protein that we have all come to rely on. But what exosomes carry depends on where they come from and where they go. Definitely one of the places of their movement is the endothelial cells that line the blood vessels. They are necessary for the growth of new blood vessels. In rats with type 2 diabetes, after two weeks of running on a wheel, and in healthy volunteers in their 50s, ATP7A levels increased after just one workout.
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Exercise did not significantly affect the weight of mice, but increased markers of endothelial function, and also affected endothelial vascular growth factor, which is required for angiogenesis. Exercise increased the amount of a powerful natural antioxidant called extracellular superoxide dismutase. This is very important in the formation of new blood vessels. (READ MORE)